| 1. | In activating these receptors , opiates prevent neurotransmitter release , thus blocking the transmission of the pain message to spinal cord neurons
鸦片类物质活化这些受体后，会抑制神经传递物的释放，因此阻断了将痛觉讯息传给脊髓神经元的过程。 |
| 2. | Our previous studies have demonstrated that capsaicin could obviously enhance the excitatory neurotransmitter release from primary afferent terminals in spinal dorsal hom
我们以往在脊髓背角的研究表明， caps能显著增加初级传入终末释放兴奋性神经递质。 |
| 3. | The present results indicate that ne depresses capsaicin - induced primary afferent terminal neurotransmitter release by activating a2 adrenergic receptor
本研究结果提示： ne通过激活初级传入终末上的a ，型肾上腺素受体，抑制caps引起的初级传入终末释放递质。 |
| 4. | ? e did not influence the spontaneous mepsc in sg . however , after pretreatment with ne , the facilitatory effect of capsaicin on neurotransmitter release from primary afferent terminals was significantly depressed
Ne对sg内自发的ilepscs无影响，但在ne预灌流后， caps增强初级传入终末神经递质释放的程度显著降低。 |
| 5. | When a neuron fires , sending an electrical signal down its axon to its tips ( presynaptic terminals ) , neurotransmitters released from vesicles cross a tiny intercellular space ( the synaptic cleft ) to receptors on the surface of a recipient , or postsynaptic , neuron
神经元放电时，电性讯号沿著轴突传至神经末梢（突触前终端） ，造成神经传递物从囊泡释放到细胞外，并通过微小的细胞间空隙（突触间隙） ，抵达接收端，也就是突触后神经元表面的受体。 |
| 6. | The changes , which may persist for a long time , can include display of increased numbers of the receptors that respond to the neurotransmitters released by nociceptors and might even include rewiring of connections and a loss of nerve cells that normally inhibit pain signaling
这种改变可能持续好长一段时间，改变的情况包括对神经传递物（由痛觉受器释放）产生反应的受体数量增加，甚至还可能是神经线路出现新的接线，以及丧失了原本抑制疼痛讯息的神经细胞。 |
| 7. | The selective action of capsaicin on primary afferent fibers that transmit nociceptive information simulates nociceptive stimulation in vivo , therefore , the ne depression of primary afferent terminal neurotransmitter release might be significant on nociceptive modulation at spinal level
Caps选择性地作用于传递伤害性信息的初级传入纤维，模拟了在体伤害性刺激，因此， ne对初级传人终末递质释放的抑制，在脊髓水平的伤害性信息调控过程中可能具有重要意义。 |
| 8. | Intracellular ca2 + serves as a ubiquitous second messenger in nerve cells . fluctuations of [ ca2 + ] termed " calcium signals " regulate a variety of intracellular events such as excitability , neurotransmitter release , synaptic plasticity , gene expression , development and differentiation
神经细胞内钙离子不仅是一种重要的第二信使，而且对神经细胞的其它功能也具有重要的调节作用，因此，有必要在完整神经细胞上准确测定钙离子的动态变化、运动方式及梯差分布。 |
| 9. | ? he pretreatment of ne together with yohimbine , an a , adrenergic receptor antagonist , had no effect on the facilitatory action of capsaicin on neurotransmitter release from primary afferent terminals . this result suggests that the a2 adrenergic receptor on primary afferent terminals might be involved in ne depression on capsaicin - induced neurotransmitter release
将飞型肾上腺素受体阻滞剂育亨宾（ yohimbine ）与ne同时预灌流后， caps对初级传入终末递质释放的易化作用不受影响，提示初级传入纤维终末上的a 。 |
| 10. | The results showed that : ? apsaicin could significantly increase the frequency of miniature excitatory postsynaptic currents ( mepscs ) , in another word , capsaicin could enhance the excitatory neurotransmitter release from primary afferent terminals . this is coincidence with the results of our previous studies
第四军医大学硕士学位论文结果表明： coys能显著增加所记录的微小兴奋性突触后电流（ miniatureexcitatorypostsympticcurrents ， inepscs ）的频率，即增强了初级传入终末释放兴奋性神经递质，这与我们以往的研究结果一致。 |
